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Signaling pathways are implicated in vascular remodeling. Among them, MAPK-ERK is activated by pro-angiogenic factors in endothelial cells (EC). MAPK-ERK is regulated by scaffold molecules which assemble components into a localized signaling complex.
By using deficient mice, we could demonstrated that KSR2 regulate EC biology and is critical in the angiogenesis process during tumor progression. We will extend and signify these findings on a set of human cancer.
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