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The gene kcnq1 codes for a potassium channel and plays a critical role in the electrophysiology of several tissues, in particular cardiac muscle. Genetic mutations of kcnq1 or its reduced expression are involved in cardiac arrhythmias associated with the risk of syncope or sudden death. The research group has recently suggested that higher order chromatin structures control kcnq1 expression during skeletal muscle differentiation. The present project aims at the identification of epigenetic mechanisms possibly involved in kcnq1 silencing, using several models of skeletal and cardiac muscle differentiation.
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Biological sciences - Biology
Number of awards per year
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International mobility required ?
Eligible destination country/ies for fellows
Eligibility of fellows: country/ies of residence
Eligibility of fellows: nationality/ies
Website of Fellowship Programme
Experienced researcher or 4-10 yrs (Post-Doc)
Employment contract with full social security
Total amount per fellowship per year
Covers travel and subsistence
Covers research costs
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