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Multiple sclerosis (MS) is a chronic autoimmune disease that is more prevalent in females. Certainly the incidence ratio of females to males appears to have increased over the last 100 years. The sex-biasis’s reason is not completely clarified, but could include genetic factors. Patients with the deletion on the CCR5 receptor have a 2-fold reduced life expectancy compared to the patients with normal genotype. This effect is more significant in females, where life expectancy is reduced by 4 times. Furthermore, in postmortem MS brains an elevated expression of CCR5 receptors was reported. The main scope of this project is to investigate the role of CCR5 in experimental models of MS and explain possible patogenetic mechanisms relative to thisreceptor
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