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Alternative splicing is the process by which a single primary transcript (pre-mRNA) can be spliced in more than one pattern to generate multiple, distinct mature mRNAs leading to expression of protein isoforms with different structural and functional properties. AS provides a versatile mechanism to regulate many biological processes and to respond to physiological stimuli.
Recently, increasing evidences indicate the altered AS profiles of critical genes may impact on all the major aspects of cancer cell biology such as proliferation, differentiation, cell death, motility and invasion supporting a causative role of altered AS in cancer.
The long-term goal of our research is to define the role of alternative splicing (AS) in regulating tumor angiogenesis and discover novel AS isoforms expressed selectively on tumor endothelial cells in order to develop anti-angiogenic approaches to control cancer growth
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